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Established at diagnosis of a 68-year-old male with acute myeloid leukemia (AML M6) secondary to myelodysplastic syndrome (MDS, subtype refractory anemia with excess of blasts, RAEB) in 1989; Cells were described to be proliferatively responsive to GM-CSF and IL-3. Cells have been shown to synthesize haemoglobin when GM-CSF or IL-3 is substituted by erythropoietin (Epo). They have been reported to be positive for leukocyte common antigen (CD45) and some multilineage markers such as CD13, CD33 and CD34, but are negative for T- and B-cell antigens and mature myelomonocytic antigens. Due to their reaction with some monoclonal antibodies recognising erythroid and platelet glycoproteins it has been suggested that F-36P has a multilineage phenotype.

于1989年診斷1例68歲男性急性髓系白血病(AML M6)繼發(fā)于骨髓增生異常綜合征(MDS，亞型難治性貧血，母細(xì)胞過多，RAEB);細(xì)胞被描述為對GM-CSF和IL-3有增殖反應(yīng)。細(xì)胞合成血紅蛋白時(shí)GM-CSF或IL-3被紅細(xì)胞生成素(Epo)取代。據(jù)報(bào)道，它們對白細(xì)胞共同抗原(CD45)和一些多系標(biāo)志物如CD13、CD33和CD34呈陽性反應(yīng)，但對T、b細(xì)胞抗原和成熟的髓單核細(xì)胞抗原呈陰性反應(yīng)。由于其與一些識別紅細(xì)胞和血小板糖蛋白的單克隆抗體的反應(yīng)，已被認(rèn)為F-36P具有多系表型。